This study examines the role of betaine-homocysteine methyltransferase (BHMT) and betaine in liver function, demonstrating that betaine supplementation enhances BHMT activity, reduces homocysteine levels, and increases S-adenosylmethionine (SAM) production. This metabolic shift protects liver cells from oxidative stress-induced apoptosis by reducing reactive oxygen species (ROS) and caspase activation. The findings
