This study investigates the effects of nicotinamide mononucleotide (NMN) on alveolar epithelial cell (AEC) senescence. NMN supplementation in aged C57BL/6 mice (8–10 months old) and in a bleomycin-induced pulmonary fibrosis model in young mice (6–8 weeks old) demonstrated significant mitigation of age-associated physiological decline in the lungs. In vitro, NMN
